Physics

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Identification of phosphorylated amino acids in cardiac PR65A.

Identification of phosphorylated amino acids in cardiac PR65A.

figure posted on 2014-01-21

PP2A phosphatase activity and interaction of PR65A with PP2Ac is reduced by PR65A phosphorylation.

PP2A phosphatase activity and interaction of PR65A with PP2Ac is reduced by PR65A phosphorylation.

figure posted on 2014-01-21

2D-DIGE Analysis of phosphoproteins from N-PR65A (green) vs. P-PR65A (red) transfected HEK cells.

2D-DIGE Analysis of phosphoproteins from N-PR65A (green) vs. P-PR65A (red) transfected HEK cells.

figure posted on 2014-01-21

Reduced expression of phosphorylated PR65A and greater PP2A activity in systolic failing hearts.

Reduced expression of phosphorylated PR65A and greater PP2A activity in systolic failing hearts.

figure posted on 2014-01-21

Proposed defect in PP2A holoenzyme assembly by phosphorylation of PR65A.

Proposed defect in PP2A holoenzyme assembly by phosphorylation of PR65A.

figure posted on 2014-01-21

Schematic of signaling elucidated in present work.

Schematic of signaling elucidated in present work.

figure posted on 2014-01-21

Phosphoprotein spots which demonstrated 2-fold or greater abundance in HEK cells transfected with P-PR65A vs. N-PR65A.

dataset posted on 2014-01-21

Calcium Induced Regulation of Skeletal Troponin — Computational Insights from Molecular Dynamics Simulations

Calcium Induced Regulation of Skeletal Troponin — Computational Insights from Molecular Dynamics Simulations

dataset posted on 2013-03-16

Consolidation of the β-sheet upon Ca<sup>2+</sup> release. A.

Consolidation of the β-sheet upon Ca²⁺ release. A.

figure posted on 2013-03-16

Hydrophobic core and hydrophobic residue re-packing following release of Ca<sup>2+</sup>.

Hydrophobic core and hydrophobic residue re-packing following release of Ca²⁺.

figure posted on 2013-03-16

Breakage of the N<sup>2</sup>-OE<sup>12</sup> and N<sup>9</sup>-OE<sup>12</sup> ‘prong’ interaction and expansion of the Ca<sup>2+</sup> binding pocket at sites I and II.

Breakage of the N²-OE¹² and N⁹-OE¹² ‘prong’ interaction and expansion of the Ca²⁺ binding pocket at sites I and II.

figure posted on 2013-03-16

RMSF of TnC N-lobe.

RMSF of TnC N-lobe.

figure posted on 2013-03-16

Open and closed state of the N-lobe. Time dependence of TnC hydrophobic pocket opening distance (d).

Open and closed state of the N-lobe. Time dependence of TnC hydrophobic pocket opening distance (d).

figure posted on 2013-03-16

The core domain of the troponin molecule.

The core domain of the troponin molecule.

figure posted on 2013-03-16

Markers of intravascular hemolysis on admission.

dataset posted on 2013-02-21

Erythrocyte surface makers on admission.

dataset posted on 2013-02-21

Surrogate markers for extravascular hemolysis and disease severity on admission.

dataset posted on 2013-02-21

Clinical data of anemic and non-anemic patients on admission.

dataset posted on 2013-02-21

Distribution of enhanced intravascular and extravascular hemolysis across the patient groups and its correlation with reduced RPI: In figure A), a dot plot of α-HBDH vs. hemopexin concentration, both makers for IVH, at presentation is shown on the x and y axis, respectively.

Distribution of enhanced intravascular and extravascular hemolysis across the patient groups and its correlation with reduced RPI: In figure A), a dot plot of α-HBDH vs. hemopexin concentration, both makers for IVH, at presentation is shown on the x and y axis, respectively.

figure posted on 2013-02-21

Phagocytosis rate in severe malarial anemia (SMA) and mild malaria (MM) patients: The plot shows individual values of relative phagocytosis of autologous samples (patients monocytes phagocytosing autologous RBCs) and positive control samples (patients monocytes phagocytosing anti-D IgG opsonized control RBCs).

Phagocytosis rate in severe malarial anemia (SMA) and mild malaria (MM) patients: The plot shows individual values of relative phagocytosis of autologous samples (patients monocytes phagocytosing autologous RBCs) and positive control samples (patients monocytes phagocytosing anti-D IgG opsonized control RBCs).

figure posted on 2013-02-21

Study-flow chart.

Study-flow chart.

figure posted on 2013-02-21

File S1 - MicroRNA Expression Is Down-Regulated and Reorganized in Prefrontal Cortex of Depressed Suicide Subjects

File S1 - MicroRNA Expression Is Down-Regulated and Reorganized in Prefrontal Cortex of Depressed Suicide Subjects

dataset posted on 2012-03-09

Characteristics of subjects in the depressed suicide and normal control groups.

dataset posted on 2012-03-09

microRNAs down-regulated in depressed suicide as determined by individual tests of statistical significance.

dataset posted on 2012-03-09

Quantile rank analysis of miRNA expression data.

dataset posted on 2012-03-09

Levels of Dicer, Drosha and DGCR8 mRNA are similar in depressed suicide subjects and normal controls.

dataset posted on 2012-03-09

Network of microRNAs pairs in normal control and depressed groups.

Network of microRNAs pairs in normal control and depressed groups.

figure posted on 2012-03-09

Protein levels of DNMT3b, BCL2, and VEGFA in PFC of depressed suicide (n = 18) and normal control (n = 17) subjects.

Protein levels of DNMT3b, BCL2, and VEGFA in PFC of depressed suicide (n = 18) and normal control (n = 17) subjects.

figure posted on 2012-03-09

Plot of Mean vs standard deviation (SD) for normal control and depressed suicide groups.

Plot of Mean vs standard deviation (SD) for normal control and depressed suicide groups.

figure posted on 2012-03-09