Now showing items 1-3 of 3

    • Enterovirus 71 3C inhibits cytokine expression through cleavage of the TAK1/TAB1/TAB2/TAB3 complex. 

      Lei, X.; Han, N.; Xiao, X.; Jin, Q.; He, B.; Wang, J. (American Society for Microbiology, 2014-09-01)
      Enterovirus 71 (EV71) causes hand, foot, and mouth disease in young children and infants. Severe infection with EV71 can lead to various neurological complications or fatal diseases. However, the mechanism of EV71 ...
    • Herpes Simplex Virus 1 Suppresses the Function of Lung Dendritic Cells via Caveolin-1. 

      Wu, B.; Geng, S.; Bi, Y.; Liu, H.; Li, X.; Zhang, Y.; Zhou, X.; Zheng, G; He, B.; Wang, B. (American Society for Microbiology, 2015-08)
      Caveolin-1 (Cav-1), the principal structural protein of caveolae, has been implicated as a regulator of virus-host interactions. Several viruses exploit caveolae to facilitate viral infections. However, the roles of Cav-1 ...
    • Reciprocal Regulation between Enterovirus 71 and the NLRP3 Inflammasome. 

      Wang, H.; Lei, X.; Xiao, X.; Yang, C.; Lu, W.; Huang, Z.; Leng, Q.; Jin, Q; He, B.; Meng, G.; Wang, J. (Elsevier, 2015-07-07)
      Enterovirus 71 (EV71) is the major etiological agent of hand, foot, and mouth disease (HFMD). Early studies showed that EV71-infected patients with severe complications exhibited elevated plasma levels of IL-1β, indicating ...